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In RIN-m5F cells, UA increases the mRNA expression of inflammatory mediators, including chemokine (C-X-C motif) ligand 1 (CXCL-1 or KC), monocyte chemoattractant protein-1 (MCP-1), and interleukin-6 (IL-6) [30]

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next, the cross-bridge breaks down ATP and becomes energized, and the neck of the kinesin twists, and then, the other head binds to the next site on the microtubule

It can contribute to insulin resistance, a condition where your cells don't respond properly to insulin, making it much harder for your body to process sugar and much easier to store fat